Cardiac Contractile Function and Mitigates Myocardial Infarction in Rats
نویسنده
چکیده
Results: Results demonstrated that OC treatment or nicotine exposure led to increased hemorheological (blood viscosity, hematocrit and plasma viscosity), prothrombotic (plasminogen activator inhibitor-1), proinfl ammatory (Creactive protein, uric acid, neutrophil/lymphocyte ratio and platelet/lymphocyte ratio) and anti-infl ammatory (corticosterone) markers. However, these effects except that on corticosterone were normalized by concomitant OC and nicotine exposure.
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